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Relationship Between Ambient Black Carbon and Daily Mortality in Tehran, Iran: A Distributed Lag Nonlinear Time Series Analysis Publisher



Rahmatinia M1 ; Hadei M2 ; Hopke PK3, 4 ; Querol X5 ; Shahsavani A6, 7 ; Namvar Z1 ; Kermani M8
Authors
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Authors Affiliations
  1. 1. Student Research Committee, Department of Environmental Health Engineering, School of Public Health and safety, Shahid Beheshti University of Medical Sciences, Tehran, Iran
  2. 2. Department of Environmental Health Engineering, School of Public Health, Tehran University of Medical Sciences, Tehran, Iran
  3. 3. Department of Public Health Sciences, University of Rochester School of Medicine and Dentistry, Rochester, 14642, NY, United States
  4. 4. Center for Air Resources Engineering and Science, Clarkson University, Potsdam, 13699, NY, United States
  5. 5. Institute of Environmental Assessment and Water Research (IDAEA), Spanish Council for Scientific Research (CSIC), Barcelona, Spain
  6. 6. Environmental and Occupational Hazards Control Research Center, Shahid Beheshti University of Medical Sciences, Tehran, Iran
  7. 7. Department of Environmental Health Engineering, School of Public Health and Safety, Shahid Beheshti University of Medical Sciences, Tehran, Iran
  8. 8. Research Center for Environmental Health Technology, Iran University of Medical Sciences, Tehran, Iran

Source: Journal of Environmental Health Science and Engineering Published:2021


Abstract

Purpose: The aim of the present study was to investigate the effect of short-term exposure to ambient black carbon (BC) on daily cause-specific mortality, including mortality due to respiratory, cardiovascular, ischemic heart and cerebrovascular diseases in Tehran, Iran. Materials and methods: Daily non-accidental death counts, meteorological data and hourly concentrations of air pollutants from 2014 to 2017 were collected in Tehran. A distributed lag non-linear model was used to assess the association between exposure to BC and daily mortality. Results: The mean daily BC concentration during the study period was 3.96 ± 1.19 µg/m3. The results indicated that BC was significantly associated with cardiovascular, ischemic heart disease, and cerebrovascular mortality, but not with respiratory mortality. In first model, each 10 µg/m3 increase in at lag 3, lag 4 and lag 5 were associated with cardiovascular mortality in 16–65 year age group with the relative risks (RRs) of 1.17 (95 % CI: 1.02–1.33), 1.17 (95 % CI: 1.04–1.31) and 1.12 (95 % CI: 1.02–1.24), respectively. The highest mortality rate per 10 µg/m3 increase in exposure was found for ischemic heart diseases with RR of 3.98 (95 % CI: 1.04–1.81, lag 01) for 16–65 age group. Cerebrovascular mortality was associated with 10 µg/m3 increases in non-cumulative exposure with RR of 1.17 (95 % 1.009–1.35, lag 5) in the age group ≥ 65 years. In the second model for a 10 µg/m3 increase in BC, cardiovascular mortality at specific lag days (5 and 6 days) in the age group ≤ 16 years were associated with RR of 1.34 (95 % CI 1.08–1.66) and 1.35(95 % CI 1.02–1.77), respectively. Conclusions: This study in Tehran found significant effects of BC exposure on daily mortality for cardiovascular, ischemic heart disease, cerebrovascular disease. © 2021, Springer Nature Switzerland AG.
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