Tehran University of Medical Sciences

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Faculty Members have conducted research related to the Quercetin Enhanced Proliferation of Neural Stem/Progenitor Cells Through Nrf2-Proteasome Pathway
Davood Zarini
Davood Zarini

Assistant Professor of Anatomical Sciences

Department Anatomy

School of Medicine

Tehran University of Medical Sciences

All Documents
1. Benefits of Bone Marrow Mesenchymal Stem Cells Compared to Their Conditioned Medium in Valproic Acid-Induced Autism in Rats, Molecular Biology Reports (2024)
2. Correction To: Calorie Restriction Promotes Remyelination in a Cuprizone-Induced Demyelination Mouse Model of Multiple Sclerosis (Metabolic Brain Disease, (2020), 35, 7, (1211-1224), 10.1007/S11011-020-00597-0), Metabolic Brain Disease (2024)
3. Microglia/Macrophage Polarization Regulates Spontaneous Remyelination in Intermittent Cuprizone Model of Demyelination, Biochemistry and Biophysics Reports (2024)
4. Secretome of Human Amniotic Membrane Stem Cells Promote Recovery and Testicular Functions Through Modulating Sirt1/Nrf2/Tnf-Α Pathway in Mice Testicular Torsion: An Experimental Study, International Journal of Reproductive BioMedicine (2024)
5. Quercetin As a Possible Complementary Therapy in Multiple Sclerosis: Anti-Oxidative, Anti-Inflammatory and Remyelination Potential Properties, Heliyon (2023)
6. Combination Effects of Mesenchymal Stem Cells Transplantation and Anodal Transcranial Direct Current Stimulation on a Cuprizone-Induced Mouse Model of Multiple Sclerosis, Journal of Molecular Histology (2022)
7. Glial Response to Intranasal Mesenchymal Stem Cells in Intermittent Cuprizone Model of Demyelination, Neurotoxicity Research (2022)
8. Protective Features of Calorie Restriction on Cuprizone-Induced Demyelination Via Modulating Microglial Phenotype, Journal of Chemical Neuroanatomy (2021)
9. Calorie Restriction Promotes Remyelination in a Cuprizone-Induced Demyelination Mouse Model of Multiple Sclerosis, Metabolic Brain Disease (2020)